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[We found that two of the HDACis tested, SAHA and ROMI, but not VPA, indeed inhibit HR repair and that RAD51, BARD1, and FANCD2 represent key proteins whose inhibition is required for HDACi-mediated therapy with PARP inhibition in TNBC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine.
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