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[Podocyte-specific genetic ablation of XBP1 or inducible expression of ATF6 in mice aggravates DN. sXBP1 lies downstream of insulin signalling and attenuating podocyte insulin signalling by genetic ablation of the insulin receptor or the regulatory subunits phosphatidylinositol 3-kinase (PI3K) p85? or p85? impairs sXBP1 nuclear translocation and exacerbates DN.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine.
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